Ibogaine+Effects

= **Ibogaine: Effects Profile** =

From anecdotal reports, it appears that memories are relived in the minds eye, but without the intensity of emotion they experienced when the events occurred, allowing the individual to view them objectively (Naranjo, 1974; Alper et al., 1999). The objectivity allows for greater neuroplasticity of memories by both the subjects and the clinician, and therefore this phenomenon has been sighted as a potentially valuable tool in psychotherapy (Naranjo, 1967, 1974). While ibogaine has similar traits as hallucinogens, it is not a hallucinogen or psychomimetic (Luciano, 1998; Goutarel, Gollnhofer, and Sillans 1993; Popik and Glick, 1996). It is suggested that it be defined as oneirogenic, due to the "waking dream" state it induces, from the Greek, meaning "dream creator" (Naranjo, 1974; Goutarel, Gollnhofer, and Sillans, 1993).

In addition to ibogaine's psychological effects, it elicits a number of physical effects, which include tremor, light sensitivity, nausea and vomiting, ataxia, and dystonia (Lotsof, 1994; Glick, Maisonneuve, and Szumlinski, 2000). All of these effects, psychological and physical, manifest in a dose-dependent fashion (Schechter and Gordon, 1993). In light of these properties, and that the sum effects of ibogaine can last up to 24-36 hours, ibogaine is not considered to have a high potential for abuse (Popik and Glick, 1996).

An experience report (with 200 mg, orally):

"The most unpleasant symptoms were the anxiety, the extreme apprehension, and the unfamiliar mood associated with visual and bodily hallucinations. The visual hallucinations appeared only in the dark ... Dysesthesia of the extremities. a feeling of light-weightedness, and hyperacusis were other symptoms noted. Autonomic signs, such as dryness of the mouth, increased perspiration, slight pupillary dilation, and increase in pulse rate, as well as extrapyramidal syndromes (fine tremors, slight ataxia, enhanced tendon reflexes and clonus) were also present. The peak effect was reached at about 2 hours after swallowing the drug; it subsided gradually, leaving as a residue complete insomnia. No undesirable after-effects, such as exhaustion or depression occurred." (Shulgin and Shulgin, 1997)